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Our recent work used a 3Tg-AD model to show that dysregulated expression of monoacylglycerol lipase (Mgll) caused by the impaired atypical protein kinase C (aPKC) mediated CREB binding protein (CBP) phosphorylation in AD can be used as a marker to guide metformin-targeted therapy (reactivating the impaired aPKC-CBP pathway) to correct perturbed neurogenesis and spatial memory in AD (Syal et al., 2020)

Endocrine (2006) 30:1916

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Long-term biocompatibility issues arise from nanoparticles which trigger inflammatory responses together with oxidative stress and immune system activation in tissues that need no treatment [43]

Being off-kilter is another common problem that people have

CVB4 P2-C protein has sequence homologies to GAD, a T1D autoantigen, and is recognized by TCR receptors on T cells against GAD65 ( Figure 2 ) (191, 192)